Acute pancreatitis represents a local inflammatory disorder with severe systemic consequences. Significant progress in understanding the pathophysiology of acute pancreatitis has been achieved in recent years. However, there is no clear concept about initialization and propagation of the disease both in experimental models and in humans. Furthermore, reliable strategies to evaluate prognosis and perform therapy are still missing. The review focuses on mechanisms originating from acinar cells leading to a systemic inflammatory response in experimental pancreatitis.
© 2001 S. Karger AG, Basel and IAP
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Number of Print Pages : 7
Number of Figures : 2, Number of Tables : 0, Number of References : 44
Official Publication of the International Association of Pancreatology (IAP); Official Publication of the European Pancreatic Club (EPC); Official Publication of the Spanish Pancreatic Club; Official Publication of the Club Português do Pancreas; Official Publication of the Pancreatic Society of Great Britain and Ireland; Official Publication of the Associazione Italiana per lo Studio del Pancreas
Vol. 1, No. 4, Year 2001 (Cover Date: 2001)
Formerly International Journal of Pancreatology
Journal Editor: Manfred V. Singer, Mannheim; Clem W. Imrie, Glasgow
ISSN: 1424–3903 (print), 1424–3911 (Online)
For additional information: http://www.karger.com/journals/pan
Article / Publication Details
Published online: 8/24/2001
Issue release date: 2001
Number of Print Pages: 7
Number of Figures: 2
Number of Tables: 0
ISSN: 1424-3903 (Print)
eISSN: 1424-3911 (Online)
For additional information: http://www.karger.com/PAN
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